Re: Common MRI poisoning some kidney patients

Dead_Man_Walking — Mon, 20 Jul 2009 12:33:50 -0000

Thank you Wendi for your kind words of appreciation and your willingness to publish my experience on this website. I continue to find more & more information/evidence on a daily basis about this man-made disease, nephrogenic systemic fibrosis. Below will be links to data that I have found. I am still willing to send your firm a copy/cd of my research, (NSF MDL 1909 plaintiff firms use only, of course). I simulated the printing of the cd several months ago and gave up from exhaustion when reaching the letter (O) in just my medical nsf .pdf files and had already exceeded 11,000 pages. I estimate the cd contains 15,000+ nsf specific data. There has been two paralegals working with my cd for several months now and have not filtered all of the data to date.

Anyway, thank you for the kind words,
danny bailey

Dr. Jerrold Abraham internet link to his research/testimony as to the lethal build-up of gadolinium metal from "contrast" enhanced radiolgical studies in the patient population.

http://www.upstate.edu/path...

The most logical pathogenosis of gadolinium/NSF has already been described in research I found of a questionably similar disease Scleroderma. This is a link to the only article I've found to date that describes in detail the know pathogenosis of Scleroderma, which common sense says is the same pathnogenosis of gadolinium/NSF. The medical community is playing dumb, but the truth is out there.

http://www.scientificameric...

A few paragraphs from the four page discussion.

Scleroderma affects roughly one hundred thousand people in the U.S. alone; exact numbers do not exist because minor cases are often misdiagnosed or not diagnosed at all. The disease causes thickening and hardening of the skin and damages arteries, joints and internal organs such as the lungs and kidneys.
"Fibrotic component. Fibrosis is a process that follows chronic inflammation in the progression of scleroderma. Fibrotic tissue is a scar tissue, which is thick and rigid because of excess accumulation of the protein (collagen). Initially, the excess of collagen produces thick, tight skin and resultant pain and burning. This problem is easily diagnosed and requires no laboratory or special testing.

"Fibrotic changes within the lung tissue may parallel the changes in the skin, giving rise to symptoms of chronic inflammation. Using high-resolution computed tomography (CT) of the lungs, researchers can evaluate thin sections of the lungs for early signs of inflammation. (A discussion of this work, along with CT images showing the effects of scleroderma, are available online from research group at at Los Alamos National Laboratory.) Repeated testing of the lung function allows the doctor to follow the response to treatment.

"Results show that exposure to toxic oxygen products causes certain tissue molecules to break apart, but only in the presence of abnormal amount of iron, copper, zinc and other metals in the body. Fragmentation of these molecules exposes hidden parts of the molecules that the immune system has never seen. The immune system 'sees' the newly exposed parts as foreign invaders and produces the antibodies against them. This produces the symptoms of scleroderma. (actual content of article, just the font color has been highlighted, no editing has been done with the content)

(this is my comment/belief as to what is really being said as fact) This is also the most logical/probable pathogenesis of gadolinium/NSF. The medical community has conveniently/intentionally withheld/suppressed their long time knowledge of the chemical "redox" reactions at the cellular level being catalyzed by abnormal accumulations of "metals" within a patients tissue. These metal catalyzed chemical reactions produce by-products (bad oxygen's, hydrogen peroxide, nitric oxide, etc..) that are capable of damaging cells by breaking them open, which in turn damages the strands of DNA contained within the cells. When the patients auto-immune system encounter these damaged cells/DNA it no longer recognizes the damaged cells as being a normal part of the patients body. The auto-immune system the launches/triggers a full scale attack against the damaged cells, which begins the fibrosing process that science claims to understand from that point on. When the auto-immune system is triggered for any reason, it not only releases the fibroblast/cytokines/etc...from the patients bone marrow, but it ALSO releases even more gadolinium that had been stored in the bone marrow from previous gadolinium exposures.

"Clinical studies are underway. Hopkins scientists are investigating what causes the abnormal cellular metal accumulation and whether the abnormal metals cause the blood vessels to narrow. The team is also studying drugs that might prevent the autoantigens from being fragmented."

Las Alamos/Oak Ridge and many other death labs need de-commissioned/de-stroyed & de-buried in a 10' thick concrete tomb. The employee's and there regulatory agency been given one chance to apply their skills to something not lethal/productive in another closely monitored environment or be sent to prison for a threat to mankind and it's so-called civilized societies.

A link to a complete listings of the Government Accountability Office audits of the FDA (448 audit results), they are informative to say the least, evidence of serious problems with our regulatory agencies to be just plain honest. Especially the discovered flaws in the FDA's monitored clinical drug trials. Anyone can go to the to the search area on the website below and delete the acronym [FDA] and type in CDC, DHS, HHS, etc......to read the audits of any federal agency,,,,,it is our tax dollars at work and a wake-up call for the taxpayers/the people.

http://www.gao.gov/docsearc...

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Re: Common MRI poisoning some kidney patients